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Paradigm Challenge  /  Biology

Antidepressants take weeks to work because they have to wait for "middleman" cells to remodel the brain's wiring.

Scientists have long been confused by why SSRIs raise serotonin levels instantly but do not improve mood for a month. It turns out that serotonin does not act on neurons directly to fix depression. Instead, it triggers a chain reaction where astrocytes release a growth factor called FGF2, which then tells the brain to rebuild its connections. This remodeling process is slow and requires a specific sequence of biological handoffs to occur. Understanding this delay allows researchers to look for ways to skip the middleman and make treatments work much faster.

Original Paper

Astrocyte-Derived Trophic Signaling Gates Serotonin-Dependent Cortical Remodeling

Vicky Yao, Zachary Gershon, Devin Rocks, Ammar Aly, Alexandra Lenz, Jodi Gresack, Laura Chalencon, Ashna Singh, Salina Kalik, Anne Schaefer, Olga Troyanskaya, Paul Greengard, Priya Rajasethupathy, Connor Liston, Revathy Uthaiah Chottekalapanda

SSRN  ·  6690583

Serotonergic signaling is central to emotion regulation and the therapeutic effects of selective serotonin reuptake inhibitors (SSRIs). Although SSRIs rapidly elevate extracellular serotonin, clinical improvement typically emerges over weeks, indicating that delayed adaptations underlie their efficacy. While diverse brain cell types coordinate complex functions, how they interact to translate serotonergic signals into long-term plasticity remains unclear. Here, we identify a temporally ordered,